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Current concepts in arthritis research: So why would hurt my knee and you can make it better?
Osteoarthritis (OA) is one of the most common Diseases leading to disability and impaired quality of life in the Western world.
Ironically, while more effective disease-modifying therapies have developed for rheumatoid arthritis, especially within the last 10-15 years have been, even rheumatologists treat osteoarthritis with symptomatic and supportive Therapies.
As a result, the relentless progression of the disease results in performance of more than half a million joint replacements annually in the United States. While joint replacement has made enormous progress, there is still a major surgical procedure.
Risk factors for the development of OA include: genetic factors, obesity, joint injury, surgery, and the presence of associated metabolic disorders.
It is from the research that clear OA is a disease that not only the cartilage-cartilage that caps the end of the long bones and cushions the joint, but also the synovial membrane tissue along the joint as also the bones, the cartilage is destroyed.
While genetic factors play an important role in the incidence of osteoarthritis, is the damage that occurs a result a complex interaction of inflammatory mediators. These include cytokines, prostaglandins, nitric oxide, growth factors and proteases.
These substances, which of chondrocytes (cartilage cells), which are produced subject to unusual forces lead to a situation where there is premature aging and destruction of cartilage.
The production of these inflammatory proteins also contributes to inflammation of the synovium and excessive amounts of bone growth.
Current Therapies, such as guidelines by the Osteoarthritis Research Society International (OARSI) issued proposed are clear aim at relieving the symptoms. These treatments include: analgesics, non-steroidal anti-inflammatory drugs (NSAIDs), topical agents ("rubbing"), and joint injections with either corticosteroids ("cortisone") hyaluronic acid or lubricants.
The current research has to find the triggers that cause inflammation and also beginning to target specific Identify the markers could identify those patients who are at highest risk for rapid progression of the disease. These markers would also be useful in the measurement improves as newer medications can slow down can be detected disease progression in OA.
However, all these studies, if and as long as certain disease-modifying osteoarthritis drugs (DMOAD) vain – drugs that slow the rate of cartilage loss can be developed.
Drugs in the inhibition of cytokine and protease function is to show some promise, but it's still too early to say whether they have the desired effect. Examples of these drugs include matrix metalloproteinase inhibitors, drugs that interleukin-1, bisphosphonates, calcitonin, and dietary supplements like glucosamine and chondroitin.
And it can not be enough to deliver drugs, to find the slow progression of the disease.
The "Holy Grail" is still the treatment (s) the cartilage to rebuild. The type of therapy, which shows the greatest promise to date is the use of autologous stem cells. These stem cells are removed from the patient and back into the affected joint with a specific matrix to which the stem cells can adhere and grow.
Initial results are promising. For more Information about stem cell therapy for osteoarthritis of the knee, contact Arthritis and Osteoporosis Center of Maryland at (301) 694-5800.
About the Author
Nathan Wei, MD FACP FACR is a nationally known, board-certified rheumatologist. For more info:
Arthritis Treatment
and
Tendonitis Treatment Tips
USPlabs Yok3d Review (Week #1)